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Dr Shripad Bhimarao Deshpande

vidwan id: 24807
Male

,

Expertise

  • Physiology

Publications

Total Articles 96
Books 0
Proceedings 0

Publications

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Scopus

Citations 256
h-index 11

CrossRef

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Citations 88
h-index 6
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Bio

Physiology,NeuroPhysiology,Neuropharmacology,Neurotoxicology,Toxicology,Toxins,Neuropeptides,Scorpion and animal toxins,Spinal reflexes,Cardiac reflexes

Personal Details

  • Male
  • Banaras Hindu University, Ajagara
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Professor Jan 2000 – Jan 2020
Banaras Hindu University | Department of Physiology

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Co-Authors (10)

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Scholarly Work

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Scholarly Publications

Aglycemia and ischemia depress monosynaptic and polysynaptic reflexes in neonatal rat spinal cord in vitro by involving different types of 5-hydroxytryptamine receptors

Open Access
ARTICLE

Ptychodiscus brevis toxin enhances the frequency-dependent depression of the monosynaptic reflex in neonatal rat spinal cord in vitro

Open Access
ARTICLE

Aglycemia and ischemia depress spinal synaptic transmission via inhibitory systems involving NMDA receptors

Open Access
ARTICLE

Effect of ischaemia & aglycaemia on the synaptic transmission in neonatal rat spinal cord in vitro

Open Access
ARTICLE

Deprenyl blocks the aglycemia-induced depression of the synaptic transmission but not the ischemia-induced depression in neonatal rat spinal cord in vitro

Open Access
ARTICLE
Authors: Jha, Archana;Das Gupta, Shyamal;Deshpande, Shripad B.

Involvement of the GABAergic system for Ptychodiscus brevis toxin-induced depression of synaptic transmission elicited in isolated spinal cord from neonatal rats

Open Access
ARTICLE

Involvement of N-methyl-D-aspartate receptors for the Ptychodiscus brevis toxin-induced depression of monosynaptic and polysynaptic reflexes in neonatal rat spinal cord in vitro

Open Access
ARTICLE

Relative potency of synthetic analogs of Ptychodiscus brevis toxin in depressing synaptic transmission evoked in neonatal rat spinal cord in vitro

Open Access
ARTICLE
Authors: Singh, J. N.;Das Gupta, S.;Gupta, A. K.;Dube, S. N.;Deshpande, S. B.